Disease Management on Diabetes Mellitus

[Harini Vannamathi]

Hyperglycemia historically characterizes diabetes mellitus. Halki Diabetes Remedy Review  The patho-physiological processes which cause hyperglycemia include insufficient insulin, impaired blood sugar disposal (insulin resistance), and elevated production of hepatic blood sugar. Type I diabetes is the outcome of the deficiency of insulin normally caused by immunologic harm into beta cells. Several diabetes mellitus type I patients have features of resistance to insulin.

Diabetes mellitus type II is the outcome of insulin resistance, frequently linked with essential obesity, elevated production of hepatic glucose and progressive decrease in the function of beta cells. The loss of function of beta cells is not immunologic interceded.

Secondary types of diabetes may happen as an outcome of pacreatectomy (insulin- shortage state), management of glucocorticoids (although utilization of glucocortocoid may plainly unmask a predisposition in developing diabetes), hemochromatosis and rare condition like antibodies into the insulin receiver. Gestational diabetes happens during pregnancy as an outcome of glucose counter narrow production of hormone and may be more regular in patients having hereditary predisposition to build up diabetes mellitus type II.

The patho-physiologic method wherein hyperglycemia adds to the problem of diabetes mellitus is not recognized yet. But the following are some facts are to be considered. Hyperglycemia enhances sorbitol build up in tissues and was invoked as device for retinopathy and also neuropathy. It also increases the absorption of protein kinase C B (PKCB) in retina, which is linked with improved absorption of vascular endothelial cell growth factor.

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